Please use this identifier to cite or link to this item: https://repositorio.uca.edu.ar/handle/123456789/11312
Título : Partial reversal of striatal damage by palmitoylethanolamide administration following perinatal asphyxia
Autor : Udovin, Lucas Daniel 
Kobiec, Tamara 
Herrera, María Inés 
Toro-Urrego, Nicolás 
Kölliker Frers, Rodolfo 
Kusnier, Carlos Federico 
Ramos Hryb, Ana Belén 
Luaces, Juan Pablo 
Otero Losada, Matilde 
Capani, Francisco 
Palabras clave : ASFIXIA PERINATALNEURONASNEUROPSICOLOGIANEURODEGENERACIONNEUROPROTECCIONPALMITOILETANOLAMIDA
Fecha de publicación : 2020
Editorial : Stefano Comai. Universidad de Padua, Italia
Cita : Udovin, L. D., Kobiec, T., Herrera, M. I., Toro-Urrego, N., Kölliker-Frers, R. A., Kusnier, C. F., Ramos Hryb, A. B., Luaces, J. P., Otero Losada, M., Capani, F. Partial reversal of striatal damage by palmitoylethanolamide administration following perinatal asphyxia [en línea] Frontiers in neuroscience. 2020. Doi: 10.3389/fnins.2019.01345. Disponible en: https://repositorio.uca.edu.ar/handle/123456789/11312
Proyecto: Neuroprotección en asfixia perinatal. Una aproximación traslacional 
Resumen : Abstract: Perinatal asphyxia (PA) is a clinical condition brought by a birth temporary oxygen deprivation associated with long-term damage in the corpus striatum, one of the most compromised brain areas. Palmitoylethanolamide (PEA) is a neuromodulator well known for its protective effects in brain injury models, including PA, albeit not deeply studied regarding its particular effects in the corpus striatum following PA. Using Bjelke et al. (1991) PA model, full-term pregnant rats were decapitated, and uterus horns were placed in a water bath at 37 C for 19 min.
URI : https://repositorio.uca.edu.ar/handle/123456789/11312
ISSN : 1662-453X (on line)
Disciplina: PSICOLOGIA
DOI: 10.3389/fnins.2019.01345
Derechos: Acceso abierto
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