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Título : | Partial reversal of striatal damage by palmitoylethanolamide administration following perinatal asphyxia | Autor : | Udovin, Lucas Daniel Kobiec, Tamara Herrera, María Inés Toro-Urrego, Nicolás Kölliker Frers, Rodolfo Kusnier, Carlos Federico Ramos Hryb, Ana Belén Luaces, Juan Pablo Otero Losada, Matilde Capani, Francisco |
Palabras clave : | ASFIXIA PERINATAL; NEURONAS; NEUROPSICOLOGIA; NEURODEGENERACION; NEUROPROTECCION; PALMITOILETANOLAMIDA | Fecha de publicación : | 2020 | Editorial : | Stefano Comai. Universidad de Padua, Italia | Cita : | Udovin, L. D., Kobiec, T., Herrera, M. I., Toro-Urrego, N., Kölliker-Frers, R. A., Kusnier, C. F., Ramos Hryb, A. B., Luaces, J. P., Otero Losada, M., Capani, F. Partial reversal of striatal damage by palmitoylethanolamide administration following perinatal asphyxia [en línea] Frontiers in neuroscience. 2020. Doi: 10.3389/fnins.2019.01345. Disponible en: https://repositorio.uca.edu.ar/handle/123456789/11312 | Proyecto: | Neuroprotección en asfixia perinatal. Una aproximación traslacional | Resumen : | Abstract: Perinatal asphyxia (PA) is a clinical condition brought by a birth temporary oxygen deprivation associated with long-term damage in the corpus striatum, one of the most compromised brain areas. Palmitoylethanolamide (PEA) is a neuromodulator well known for its protective effects in brain injury models, including PA, albeit not deeply studied regarding its particular effects in the corpus striatum following PA. Using Bjelke et al. (1991) PA model, full-term pregnant rats were decapitated, and uterus horns were placed in a water bath at 37 C for 19 min. | URI : | https://repositorio.uca.edu.ar/handle/123456789/11312 | ISSN : | 1662-453X (on line) | Disciplina: | PSICOLOGIA | DOI: | 10.3389/fnins.2019.01345 | Derechos: | Acceso abierto |
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