Please use this identifier to cite or link to this item: https://repositorio.uca.edu.ar/handle/123456789/6205
Título : Could perinatal asphyxia induce a synaptopathy? : new highlights from an experimental model
Autor : Herrera, María I. 
Otero Losada, Matilde 
Udovin, Lucas Daniel 
Kusnier, Carlos 
Kölliker Frers, Rodolfo 
Souza, Wanderley de 
Capani, Francisco 
Palabras clave : ASFIXIA NEONATALSISTEMA NERVIOSOTRASTORNOS DEL NEURODESARROLLOPSICOPATOLOGIANEUROPSICOLOGIABEBESPSICOLOGIASINAPTOPATIA
Fecha de publicación : 2017
Cita : Herrera, M.I., Otero Losada, M., Udoven, L.D., Kusnler, C., Kölliker Frers, R., Souza, W., Capani, F. (2017). Could perinatal asphyxia induce a synaptopathy? : new highlights from an experimental model [en línea]. Neural plasticity. Disponible en: https://repositorio.uca.edu.ar/handle/123456789/6205
Resumen : Abstract: Birth asphyxia also termed perinatal asphyxia is an obstetric complication that strongly affects brain structure and function. Central nervous system is highly susceptible to oxidative damage caused by perinatal asphyxia while activation and maturity of the proper pathways are relevant to avoiding abnormal neural development. Perinatal asphyxia is associated with highmorbimortality in term and preterm neonates. Although several studies have demonstrated a variety of biochemical and molecular pathways involved in perinatal asphyxia physiopathology, little is known about the synaptic alterations induced by perinatal asphyxia. Nearly 25% of the newborns who survive perinatal asphyxia develop neurological disorders such as cerebral palsy and certain neurodevelopmental and learning disabilities where synaptic connectivity disturbances may be involved. Accordingly, here we review and discuss the association of possible synaptic dysfunctionwith perinatal asphyxia on the basis of updated evidence froman experimental model
URI : https://repositorio.uca.edu.ar/handle/123456789/6205
Disciplina: PSICOLOGIA
Derechos: Acceso Abierto
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