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Campo DC | Valor | Lengua/Idioma |
---|---|---|
dc.contributor.author | Cardinali, Daniel Pedro | es |
dc.contributor.author | Scacchi Bernasconi, Pablo A. | es |
dc.contributor.author | Reynoso, Roxana | es |
dc.contributor.author | Reyes Toso, Carlos F. | es |
dc.contributor.author | Scacchi, Pablo | es |
dc.date.accessioned | 2019-05-02T14:01:13Z | - |
dc.date.available | 2019-05-02T14:01:13Z | - |
dc.date.issued | 2013 | - |
dc.identifier.citation | Cardinali, D. P., et al. Melatonin may curtail the metabolic syndrome : studies on initial and fully established frucose-induced metabolic syndrome in rats [en línea]. International journal of molecular sciences. 2013, 14. doi:10.3390/ijms14022502. Disponible en: https://repositorio.uca.edu.ar/handle/123456789/1639 | es |
dc.identifier.issn | 1661-6596 (impreso) | - |
dc.identifier.issn | 1422-0067 (online) | - |
dc.identifier.uri | https://repositorio.uca.edu.ar/handle/123456789/1639 | - |
dc.description.abstract | Abstract: To examine the effect of melatonin given to rats simultaneously with fructose on initial and fully developed metabolic syndrome, male Wistar rats had free access to chow and 5% or 10% fructose drinking solution for 8 weeks. As compared to controls, systolic blood pressure augmented significantly under both treatments whereas excessive body weight was seen in rats receiving the 10% fructose only. Rats drinking 5% fructose showed a greater tolerance to a glucose load while rats having access to a 10% fructose drinking solution exhibited the expected impaired glucose tolerance found in the metabolic syndrome. Circulating triglyceride and low density lipoproteins-cholesterol (LDL-c) concentration augmented significantly in rats showing a fully developed metabolic syndrome only, while high blood cholesterol levels were found at both stages examined. Melatonin (25 μg/mL drinking solution) counteracted the changes in body weight and systolic blood pressure found in rats administered with fructose. Melatonin decreased the abnormal hyperglycemia seen after a glucose load in 10% fructose-treated rats but it did not modify the greater tolerance to glucose observed in animals drinking 5% fructose. Melatonin also counteracted the changes in plasma LDL-c, triglyceride and cholesterol levels and decreased plasma uric acid levels. The results underline a possible therapeutical role of melatonin in the metabolic syndrome, both at initial and established phases | es |
dc.format | application/pdf | es |
dc.language | eng | es |
dc.language.iso | eng | es |
dc.publisher | Multidisciplinary Digital Publishing Institute | es |
dc.rights | Acceso Abierto | es |
dc.rights.uri | https://creativecommons.org/licenses/by-nc-sa/4.0/ | es |
dc.source | International journal of molecular sciences. 2013, 14 | es |
dc.subject | SINDROME METABOLICO | es |
dc.subject | MELATONINA | es |
dc.subject | HIPERTENSION | es |
dc.subject | FRUCTOSA | es |
dc.subject | DISLIPIDEMIA | es |
dc.subject | OBESIDAD | es |
dc.subject | TOLERANCIA A LA GLUCOSA | es |
dc.title | Melatonin may curtail the metabolic syndrome : studies on initial and fully established fructose-induced metabolic syndrome in rats | es |
dc.type | Artículo | es |
dc.identifier.doi | 10.3390/ijms14022502 | - |
uca.path | Facultad de Ciencias Médicas|Departamento de Docencia e Investigación | es |
uca.disciplina | MEDICINA | es |
uca.filename | /home/data-uca-generic/folder_generic/IIBiomedicas/melatonin-may-curtail-metabolic-syndrome/metadata.xml | es |
uca.issnrd | 1 | es |
uca.affiliation | Fil: Cardinali, Daniel Pedro. Pontificia Universidad Católica Argentina. Facultad de Medicina. Departamento de Docencia e Investigación; Argentina | es |
uca.affiliation | Fil: Scacchi Bernasconi, Pablo A. Pontificia Universidad Católica Argentina. Facultad de Medicina. Departamento de Docencia e Investigación; Argentina | es |
uca.affiliation | Fil: Reynoso, Roxana. Pontificia Universidad Católica Argentina. Facultad de Medicina. Departamento de Docencia e Investigación; Argentina | es |
uca.affiliation | Fil: Reynoso, Roxana. Universidad de Buenos Aires. Facultad de Medicina. Departamento de Fisiología; Argentina | es |
uca.affiliation | Fil: Reyes Toso, Carlos F. Universidad de Buenos Aires. Facultad de Medicina. Departamento de Fisiología; Argentina | es |
uca.affiliation | Fil: Scacchi, Pablo. Pontificia Universidad Católica Argentina. Facultad de Medicina. Departamento de Docencia e Investigación; Argentina | es |
uca.version | publishedVersion | es |
item.fulltext | With Fulltext | - |
item.grantfulltext | open | - |
item.languageiso639-1 | en | - |
crisitem.author.dept | Consejo Nacional de Investigaciones Científicas y Técnicas | - |
crisitem.author.dept | Instituto de Investigaciones Biomédicas - BIOMED | - |
crisitem.author.dept | Facultad de Ciencias Médicas | - |
crisitem.author.orcid | 0000-0002-0813-9088 | - |
crisitem.author.parentorg | Facultad de Ciencias Médicas | - |
crisitem.author.parentorg | Pontificia Universidad Católica Argentina | - |
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